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It's the ECG's that george rejects that makes George's ECG's the best.
Intraventricular Conduction Delay (IVCD)
Report:Sinus rhythm 60/min Intraventricular conduction delay (defect, IVCD) Diffuse nonspecific ST/T changes Prominent U wave Comment:Characteristically, the patient’s potassium fell from 3.5 to 1.8 mEq/L following absorption of hydroxychloroquine; so
Negative T & U Waves
Report:Sinus rhythm 78/min Diffuse nonspecific ST/T changes Prominent U waves ?hypokalæmia Comment:U wave polarity follows that of the preceding T wave, except in ischæmia, where isolated U negativity may be a marker of critical LAD artery lesions. In
Hypokalæmia: Large TU Waves
Report: Sinus rhythm Large TU waves consistent with hypokalæmia Comment: Normally, T waves become flat and the U waves increase in amplitude with hypokalæmia; occasionally, T waves remain upright and merge with U waves to produce striking TU waves like
Faulty Calibration
Report: Sinus rhythm Faulty standardisation (upper left-hand corner) Sloping ST segment depression probably due to faulty standardisation Comment: The repeat trace in the CCU was completely normal (Fig 201a below). Instead of receiving an apology and
The P-on-U Effect
HypokalæmiaReport: Sinus tachycardia 135/min Non-specific ST/T changes Prominent U waves Consistent with hypokalæmia Comment: As stated earlier, the pun is Schamroth’s174. This second example is to reward the fast learners. The large U wave is superi
U Waves
Report: Sinus rhythm Non-specific T wave changes Prominent U waves[!xe "U wave:hypokalæmia" \b!] Comment: The patient became hypokalæmic following hæmodialysis; she also had chronic hypocalcæmia. It is very difficult to measure the true QT interval in
Hypokalæmia
Report:Sinus rhythm. Borderline left axis deviation -30o. Prominent repolarisation changes suggestive of hypokalæmia. Comment:The patient had severe metabolic acidosis and marked, paralysing hypokalæmia (K 1.7 mEq/L) No cause was found during her 4-day
VEBs & U Waves: Hypokalæmia
ReportSinus rhythm. Sinus arrhythmia. VEBs, bigeminy. Prominent U waves consistent with hypokalæmia. Comment:The ECG monitor alarmed at the heart rate 34/min. This need not be merely spurious bradycardia, a mistake in the first place, due to negative
Hypokalæmia, pre-VF
Report:Sinus rhythm Right atrial abnormality Poor R wave progression Probable left ventricular hypertrophy Nonspecific ST/T changes Prolonged QT interval ? large U waves Somatic tremor Comment:Potassium was 2.0 mEq/L and the pH 7.66. Most patients
Gitelman’s Syndrome: Hypokalæmia
Report:Sinus rhythm 85/min Right axis deviation +140o Right bundle branch block Large TU waves c/w hypokalæmia Comment:This is, almost, a P-on-U phenomenon (no pun intended), as Schamroth put it11. The diagnosis can only be made, as reported, in a con